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Do Antiperspirants Cause Breast Cancer?

Breast cancer and cyst formation are at epidemic proportions. At what point can we say, conclusively, that aluminum in antiperspirants is causal? The evidence grows stronger and stronger, largely due to the labors of one dedicated scientist, but she states that absolute proof does not yet exist. So, how much proof do you require before deciding it’s better to be safe than sorry?

by Heidi Stevenson

Antiperspirants: Cause of Breast Cancer? discussed some of Keele Conference scientist Philippa Darbre’s work on the likelihood of a relationship between aluminum in antiperspirants and breast cancer, in particular locations of aluminum in breast tissue and aluminum as an estrogen mimic. This article follows her work into other areas as the noose tightens around antiperspirant aluminum as a serious risk in breast diseases.

As previously noted in Antiperspirants: Cause of Breast Cancer?, Dr. Philippa Darbre’s latest presentation at the Keele Conference was on research demonstrating the ability of aluminum to cause changes in proteins that may lead to cancer metastasis, which can turn a benign tumor into a malignant metastatic cancer. Here is information she’s elicited in earlier work that has led to the study on aluminum’s potential to cause breast cancer metastasis.

Aluminum’s Interference with Iron Homeostasis

As noted in the previous report, aluminum is found in the breasts of women with breast cancer and it’s more prevalent in the region where the majority of cancers occur. Though too much iron is toxic, some is required for health. Transferrin binds iron in body fluids, and ferritin holds onto iron in cells, releasing it as needed. Aluminum can interfere with the function of ferritin and transferrin, thus destabilizing iron homeostasis.

Dr. Darbre worked with Professor Ferdinando Mannello on aluminum content in nipple aspirate fluid (which is exactly what it sounds like). They and the rest of their team obtained nipple aspirate fluid from both women with and without cancer, and examined it for levels of ferritin and transferrin.^[1] They withdrew nipple aspirate fluid (NAF) from the breasts of 16 women without cancer and 19 with cancer. Here’s a table of their results:

Table - Al in Nipple Aspirate Fluid First, notice that the amount of aluminum in NAF from cancerous breasts is more than double that of non-cancerous breasts! (268.4 µg/L / 131.3 µg/L) Notice also that ferritin is about five times higher in cancerous breasts (280.0 µg/L / 55.5 µg/L), and transferrin is almost 3 times higher (8.3 µg/L / 28 µg/L).

Graph - Cancer vs No Cancer, Aluminum, Ferritin, and Transferrin Milk levels of aluminum are greater than serum levels. That’s very important for nursing mothers to know, so that they can try to find ways ot lessening their aluminum load.

You can see the relative comparison in aluminum, ferritin, and transferrin between cancerous and noncancerous breasts in the bar graph. (Note that a separate graph is used for transferrin because there’s so much less of it that the difference wouldn’t be obvious on the scale used for aluminum and ferritin.)

Scatter Graphs - Al, Ferritin, and Transferrin The scattergraphs to the left definitively demonstrate a genuine connection between aluminum and both ferritin and transferrin. To see if a correlation exists between two sets of points, simply plotting them on a graph is fairly straightforward. If there is a relationship, then you can generally see the points arranging around a line, as you can clearly see is true for aluminum and ferritin in cancer patients (top left graph), and also for aluminum and transferrin in cancer patients.

However, if you look at the bottom two graphs, it’s quite clear that there is no correlation between aluminum and either ferritin or transferrin in non-cancerous patients.

Aluminum’s Association with Breast Cysts

A common problem in women is gross cystic breast disease. Though generally considered benign, it can be quite painful and results in a huge number of doctor visits for treatment, usually by aspiration or, in extreme cases, surgical removal. Occasionally, these cysts can mimic cancer, resulting in testing, biopsies, and worry. These cysts are divided into two types: Type I are more likely to develop into more severe pathologies—meaning cancer—than type II cysts. Women who suffer from type I cysts are more likely to develop them frequently and in large numbers.

Philippa Darbre investigated the potential association between antiperspirant aluminum and gross cystic breast disease.^[2] The report on the study states:

On the basis that antiperspirant formulations are designed to block apocrine sweat ducts of the axilla, and breast cysts result from blocked breast ducts, it has recently been hypothesized that breast cysts might also arise from antiperspirant use.

Table - Al Concentrations in Breast Cyst Fluid Darbre and associates examined breast cyst fluid (BCF) from 48 samples, 27 of type I and 21 of type II. These results were compared with samples from blood sera and milk. Result averages are shown in this table published from the study.

The average amount of aluminum found in breast cyst fluid (BCF) was 90.0 µg/L, with 150.0 µg/L in type I cysts (the more cancer-prone type) and 32.0 µg/L in type II cysts. Notice that the amounts of aluminum found in blood sera, healthy and non-cancerous breast tissue in cancerous breasts (GCBD-matched) was significantly lower than found in either type I or II cyst.

Aluminum levels found in general milk samples and specific types of milk (colostrum, intermediate, and mature) are much higher than in healthy or non-cancerous tissues. This might be of serious concern to nursing mothers, as it indicates high levels of aluminum in their breast milk, irregardless of whether they suffer from cystic breast disease.

Graph - Al Found in Type I and II Breast Cysts The amount of aluminum found in both cyst types is dramatically greater than in normal tissues or milk, but particularly notable is that the more cancer-prone breast cysts have extremely high levels of aluminum, averaging about 150.0 µg/L. The graph to the left makes this easy to see. Note, also, that there is no overlap between the lowest amount of aluminum found in type I breast cysts and the greatest amount found in type II breast cysts. Also note that, in the table above, the p-value is less than .0001, indicating that there is almost no likelihood that the results were obtained by chance.

Do Antiperspirants Cause Breast Cancer?

Writing for Elsevier’s Encyclopedia of Environmental Health 1st Edition, Dr. Philippa Darbre discusses the fact that more and more breast cancer is found in the upper outer quadrant (UOQ) of the breast.^[3] Although it had been suggested that this might be a result of weight gains, the facts don’t fit that idea. She wrote:

Numerous clinical studies dating back to the 1920s have shown that the UOQ of the breast is the most frequent site of carcinoma, but strangely the relative proportion
of breast cancer in the UOQ appears to rise chronologically with the year of publication.

She presented the following graphs to help explain:

The relative incidence of breast cancer in the upper outer quadrant (UOQ) of the breast, as reported in the scientific
literature, appears to rise chronologically with year of publication from 1928 to 1994. Using a single study population of England and Wales, the relative percentage of UOQ breast cancer has risen annually on a linear basis between 1979 and 2000, and in independent records in Scotland, a similar trend was noted between 1980 and 2001.

These graphs are pointing out that the relative rate of increase in cancer of the upper outer quadrant keeps increasing at a linear rate, year after year, in comparison to all other breast cancer locations. The top bar shows this in terms of publications that have reported on the phenomenon since 1928, when UOQ cancers were 30.9% of all breast cancers, but had reached 60.7% in 1994. Fat cannot be to blame for the increase in UOQ cancers, because it cannot explain why it would increase so consistently relative to other breast cancer locations.

There are certainly other carcinogenic chemicals in the environment that could be playing a factor in breast cancer increases. Dr. Darbre discusses them, including parabens, bisphenol A, phthalates, polycyclic musks, triclosan, and ultraviolet light filters (sun screen lotions and cosmetics). However, the amount of aluminum found in antiperspirants is very high, and it’s applied on and next to breast tissue and in the area of the greatest increase in cancer, the upper outer quadrant.

Furthermore, aluminum in antiperspirants is left on the skin and is absorbed through the skin. Antiperspirants work by combining with wastes in perspiration ducts, thus plugging them, limiting detoxification capability.

Dr. Darbre’s still unpublished study presented at the last Keele Conference demonstrates that aluminum has the ability to create changes in breast tumors, making them metastasize. It stands in addition to an already extensive body of work building the case of antiperspirants’ adverse effects. She is, though, the first to clarify that a cause and effect connection between antiperspirants and breast cancer has not yet been demonstrated. Nonetheless, her research keeps drawing the noose tighter.

The question you must answer is: How absolute must the proof be before you want to take action to prevent potential harm from a product, especially one that is not necessary for life? It’s quite obvious that our regulatory agencies are not moving to protect us, though the odds of a woman developing breast cancer are now about 1 in 8—and more and more men are also developing it.

Surely the precautionary principle says that the information now available on an association between antiperspirant aluminum and breast cancer is far more than required to stop using antiperspirants until shown to be safe. It is, though, up to you to decide how much risk you’re willing to accept.

Sources:

Analysis of aluminium content and iron homeostasis in nipple aspirate fluids from healthy women and breast cancer-affected patients; Journal of Applied Toxicology; Ferdinando Mannello, Gaetana A. Tonti, Virginia Medda, Patrizia Simone, Philippa D. Darbre; DOI: 10.1002/jat.1641
Concentration of aluminium in breast cyst fluids collected from women affected by gross cystic breast disease; Journal of Applied Toxicology; Ferdinando Mannello,a Gaetana A. Tonti and Philippa D. Darbre; DOI: 10.1002/jat.1384
Personal Care Products and Breast Cancer; Elsevier; Encyclopedia of Environmental Health 1st Edition; P. Darbre;

Tagged aluminum antiperspirants, aluminum breast cancer, aluminum breast cysts, Analysis of aluminium content and iron homeostasis in nipple aspirate fluids from healthy women and breast cancer-affected patients, antiperspirants breast cancer, antiperspirants breast cysts, Concentration of aluminium in breast cyst fluids collected from women affected by gross cystic breast disease, darbre antiperspirant research, darbre cancer research, do antiperspirants cause breast cancer, does aluminum cause breast cancer, Personal Care Products and Breast Cancer, philippa darbre, precautionary principle, science, scientist philippa darbre

No Historical Benefit in Vaccines: Polish Study

The evidence continues to mount. That vaccines are doing a great deal of harm is well beyond denying. Worse, though, the evidence that vaccines have had little or no effect on infectious diseases is clear, as documented in new graphs. The precautionary principle, which is enshrined in a UN directive, should have been implemented before vaccines were ever routinely injected.

by Heidi Stevenson

Yesterday’s article, Vaccines Do Irreparable Harm: Study from Poland, documents the revealing information brought out by Polish scientists’ review of the scientific literature on vaccination’s adverse effects and immune system effects. Today, the rest of the study covering neurological symptoms following vaccination and a history of vaccines demonstrating little benefit is reviewed.

Post-Vaccine Neurological Complications

The authors focused primarily on the preservative thimerosal’s active ingredient, mercury, in their discussion of vaccine-induced neurological damages. They noted that the percentage of people who have allergic reactions to thimerosal has been calculated at a low of 13% in the Netherlands to a high of 21% in Austria. Allergic responses to mercury are often initially triggered by vaccine injections. It’s noted for toxicity to the heart, liver, kidneys, and nervous system, along with being a carcinogen.

Readers of Gaia Health are probably aware that much current research on vaccine adjuvants indicates that aluminum may be the greatest concern. The reduced mercury load in the United States’ vaccines did reduce the autism burden for a short time in the US. However, a great number of vaccines was added to the schedule shortly after, and many of them included aluminum as an adjuvant, thus increasing the autism burden.

Over the last 20 years, neurological conditions such as autism, ADHD, mental retardation, epilepsy have increased many times over across the world. The authors wrote:

[F]rom the 1990s new vaccines for infants containing thimerosal began to be used in America. In the DTP, Hib and Hep B vaccines, children received a dose of 62.5 ug of mercury, which is 125-fold more than the dose considered safe (0.1ug/kg/day). These reports were the reason that Scandinavian countries already prohibited the use of mercury in 1990

The deaths of 19 infants of SIDS (sudden infant death syndrome) shortly after vaccination with two hexavalent vaccines were described in a paper. Those authors suggested that, though it hadn’t been proven that vaccines had caused their deaths, “it is a signal that brings to attention the need to monitor the course of vaccination and its complications”.

Epidemiological research has shown a direct relationship between increasing doses of thimerosal and the rate of autism in the US from the late ’80s through the mid ’90s. They found a correlation between the number of measles-containing vaccines and autism prevalence during the ’80s. The same researchers also found odds ratios that were statistically significant, indicating that increasing doses of mercury from vaccines with thimerosal correlate to increasing rates of autism.

Several of Poland’s childhood vaccines contain mercury, including:

Euvax: Hepatitis B, made by LG Life Sciences in Korea – 50µg/dose
DT: Diphtheria & Tetanus, made by Bio-med in Krakow, Poland – 50µg/dose
Td: made by Biomed, Krakow, Poland – 50µg/dose
DTP: Diphtheria, Tetanus, & Pertussis; made by Biomed, Krakow, Poland – 50µg/dose
D,d: Diphtheria, made by Biomed, Krakow, Poland – 50µg/dose
TT: Typhus, made by Biomed, Krakow, Poland – 50µg/dose

Note: Not included in the review is the fact that Poland’s vaccination schedule is mandatory. Notice that all but one of the vaccines listed above are made in Poland. It’s one way to assure profits for the home-grown pharmaceutical industry!

The authors discuss particular concern over the DTwP (Diphtheria, Tetanus, & wholecell Pertussis) vaccine. It is known to result in cytomegalovirus, which can result in severe neurological sequelae, including coma, hypotonia, seizures, and necrotizing encephalopathy. They state that replacing DTwP with DTaP (acellular) vaccine, and OPV (oral polio vaccine) with IPV (inactivated polio vaccine), can reduce the risk of fever after the first dose by 99%, the risk of inconsolable crying by 87%, and the risk of hypotonic-hyporesponsive episodes by 56%.

Other complications found with vaccines on the Polish schedule include;

Guillain-Barré syndrome after influenza, hepatitis, meningitis C, polio and HPV vaccines
Transverse myelitis after cholera, typhoid, and polio vaccines
Influenza, flaccid paralysis, meningitis, encephalitis, convulsions and facial palsy after oral polio vaccine
Rapid progression of retinopathy in premature infants after BCG vaccination

Vaccination History

In 1996, Poland introduced a monitoring system to record adverse effects of vaccinations. A study done by Zielinski of adverse effect reports from 1996-2000, stated:

[T[hey met astonishing examples of ignorance of the medical staff, including specialists, in their duty to report the AEFI [adverse effects following inoculation].

Unfortunately, there is no laboratory test to confirm a cause and effect association between an adverse effect and vaccination. While that makes it difficult, the authors of this review report:

[T]here are also no reports in the literature (except those listed above) of research work in immunology in the context of reactions following vaccination. It should also be noted that in more developed countries, there is little incentive for doing appropriate follow-up and laboratory tests on individuals who suffered serious adverse reactions following vaccinations.. The reason for such oversight is likely due to the fact that historically, vaccines have not been viewed as inherently toxic by the regulatory agencies.

So, the upshot is that there is precious little research into the adverse effects of vaccination. As the authors state:

The resulting lack of evidence of causality between vaccinations and serious adverse outcomes has thus been filled with an assumption that vaccines are safe.

However, even if adverse effects were minor or rare, it is still necessary to demonstrate that vaccinations are beneficial. If they are not, then no amount of risk in vaccination is acceptable.

This is where historical records of infectious disease cases and deaths come in handy. Take, for example, these two graphs on the number of cases of tuberculosis (TB) and number of deaths from it:

They are reports from Germany showing, on the left, the number of TB mortalities from 1956-1988, and on the right, the number of reported cases from 1949-1987. The BCG vaccine against TB was introduced in 1970. (The area in the rectangles represents the total number of vaccinations given during the decade from 1970-1980.) It matters not whether you take a quick look or a long one, it’s obvious that the TB vaccination had absolutely nothing to do with the reduction in either cases of TB or deaths from it.

To the right is a graph of the pertussis (whooping cough) mortality rate from 1946-1987 in Germany, with the times that the original pertussis vaccine was introduced and the diphtheria-pertussis-tetanus vaccine took its place. As can be seen, the death rate from pertussis stayed on approximately the same downward trajectory it had been on at the time of vaccine implementation.

The graph to the left shows the pertussis mortality rate in Switzerland from 1910-1980. The shaded area (at the top right of the graph) shows the introductory date of the pertussis vaccine in 1944, and shows how that vaccine’s coverage increased to nearly 100% by 1980. However, it is not possible to see any change in the trajectory of reduced pertussis deaths.

The next graph, shown to the right, shows the number of diphtheria deaths in Germany from 1920-1987. Notice that the diphtheria vaccine was introduced in 1925, with an enormous increase in the rate of disease rate!

On looking at these graphs, how seriously can we take claims that vaccines have saved lives? We can’t see any improvement in either mortality or number of cases before and after vaccinations were initiated—and in the case of diphtheria, it certainly looks like vaccinating aggravated the rate of disease!

Conclusion

The review refers to a report in the 2002 Lancet that states:

[T]he weight of evidence collectively suggests that personal and environmental hygiene reduces the spread of infection …

… Thus results from this review demonstrate that there is a continued, measurable, positive effect of personal and community hygiene on infections.

Our review authors state that the same article in the Lancet also showed that mortality from infectious diseases in general had decreased to “nearly negligible levels long before introduction of universal vaccination practices.”

They point out that the vaccine schedule has increased dramatically since the time of these studies, and antigens are being injected again and again. They then state:

Doctors and researchers point to the worsening state of health of the child population since the 1960s, which coincided with increasingly introduced vaccinations. Allergic diseases, including asthma, autoimmune diseases, diabetes and many neurological dysfunctions – difficulty in learning, ADD (attention deficit disorder), ADHD (attention deficit hyperactivity disorder), seizures, and autism – are chronic conditions, to which attention has been brought.

So, for no apparent benefit in reducing infectious diseases, vaccines are being mandated, in spite of the fact that evidence points strongly to the suggestion that vaccines are fueling the astronomically rising rates of chronic autoimmune and neurological disorders.

Of course, political realities do exist for these researchers. In spite of their powerful findings, they still don’t suggest that vaccines should be eliminated. They do, however, offer several suggestions around the idea of reducing their application. They conclude with:

It seems that it would be worthwhile to apply the precautionary principle – the ethical principle (from 1988) according to which if there is a probable, although poorly known, risk of adverse effects of new technology, it is better not to implement it rather than risk uncertain but potentially very harmful consequences.

So, we’re back to the precautionary principle, something that should have been applied decades ago. It’s difficult to imagine the massive amount of harm that’s been brought to generations of children, with each succeeding one suffering worse than the last. Will those in power ever listen to reason, or will they continue to be led around by their greed, leaving our children and future generations to suffer? Do they really believe that society can continue to function when most people are chronically ill?

Source:

Neurologic adverse events following vaccination; Progress in Health Sciences; Sienkiewicz D., Kulak W., Okurowska-Zawada B., Paszko-Patej G.

Vaccines Do Irreparable Harm: Study from Poland

The authors of this powerful new review wrote: “It is not reasonable to assume that manipulation of the immune system through an increasing number of vaccinations during critical periods of brain development will not result in adverse neurodevelopmental outcomes.” Here’s what they found.

by Heidi Stevenson

A new scientific review from Poland discusses irreparable harm done by vaccines. This review addresses the issue in terms of adverse effects, immune system effects, neurological symptoms following vaccination, and a history of vaccines demonstrating little benefit. It centers mostly on studies not often referenced in the western world, providing fresh and broad-ranging information. An honest reading of the study can leave little doubt that harm done may be extensive and often permanent.

Adverse Effects

Different systems of defining types of vaccination adverse events include dividing between immune system and nonimmune system, or classifying according to whether they’re local, general, shortly after vaccination, or develop more slowly. Regardless of how they’re distinguished, the authors state:

Reports in many Polish and foreign medical journals lead us to conclude that postvaccinal complications among children can be observed in sporadic cases and that they are disproportionate to the benefits of vaccination in the elimination of dangerous diseases in childhood.

Their study leaves little doubt that vaccines produce serious risks that are entirely out of line with any benefits gained.

They first point out that several adverse effects that occur shortly after vaccination are acknowledged by Polish law. These include:

Local reactions, including:
- local reactions after the BCG (tuberculosis) vaccine
- swelling
- lymphadenopathy
- abscess at the injection site
Postvaccinal adverse events of the central nervous system:
- encephalopathy
- febrile convulsions
- non-febrile convulsions
- paralytic poliomyelitis caused by vaccine virus
- encephalitis
- meningitis
- Guillain-Barré syndrome
Other adverse events following immunization:
- joint pain
- hypotonic-hyporesponsive episode
- fever above 39°C
- thrombocytopenia
- continuous inconsolable crying

Likely major post-vaccine neurological complications, which usually ensue more than 48 hours after vaccination, tend to be permanent nerve damage to the central nervous system, including seizures (especially if there is no increase in body temperature), hypotonic-hyporesponsive episodes, postvaccinal encephalitis, postvaccinal encephalopathy, and autism.

Immune System Effects

A neonate’s immune system does not function in the same manner as an adult’s, and it does not reach maturity until about age 3 years. The neonate’s humoral immune system—the part that functions through antibodies—comes directly from the mother. Associated with the immunoglobulin IgG that’s provided by the mother, it starts about 6 months before birth, and disappears by about 6 months after birth.

Autonomously-produced antibodies are called immunoglobulins. While the fetus starts to develop some immunoglobulin before birth, it takes time, in most instances more than a year, to reach maturity. The graph to the right shows the pace at which IgG from the mother increases and decreases, and the rate at which a neonate starts to produce its own, plus IgM, IgA, and IgD.

Because of a neonate’s existing antibodies from the mother, vaccination against some microorganisms simply does not provide any lasting protection for newborns. The authors state:

It is well established that early-life immune responses are weaker and of shorter duration than elicited in immunologically mature hosts. Consequently, vaccine efficacy in early infancy (particularly in the first 6 months of age) is limited.

They go on to state that:

[E]xperimental evidence clearly shows, that simultaneous administration of as little as two to three immune adjuvants, or repeated stimulation of the immune system by the same antigen can overcome genetic resistance to autoimmunity.

The common practice of administering more than one adjuvant at a time or repeatedly injecting the same antigen can produce autoimmune disorders.

They further point out that the toxicity of adjuvants can produce a range of adverse reactions.

The manner in which the body responds to vaccines is not well understood. It is, though, believed that the Th1 pathway is sacrificed in favor of the Th2 pathway. This is believed to lead to development of allergies in infants. Allergy, a life-threatening condition that was once rare, is now known to exist in at least 35%, possibly as many as 40%, of children. It has become one of the most serious health concerns in Europe.

The authors cite a study documenting that a natural chicken pox infection protects against asthma and atopic dermatitis in children, but the vaccine does not.

They state that:

[A]nnual vaccination against influenza has been shown to hamper the development of virus-specific CD8⁺T-cell immunity in children.

And then point out:

[I]t appears that the necessary Th1/Th2 balance is better provided by natural challenges (i.e., in a form of relatively benign childhood diseases such as chickenpox and mumps) rather than vaccination.

Recent research by Singh of the International Institute for Brain Research in the USA confirm the veracity of this statement. In the study, serum and cerebrospinal fluid (CSF) were analyzed in terms of viral and autoimmune markers in patients with autism compared with a group of healthy children

The table to the left, which the authors reprinted from the Singh study, shows differences in antibody levels they found between normal and autistic children. Notice that autistic children have a significantly higher vaccine measles virus antibody load than normal children, though the differences in mumps and rubella antibodies does not reach a significant level, and the high p-values for them bear that out.

A p-value approaching 1.0 indicates that the results could readily have been reached accidentally. (Note: HHV-6 refers to human herpes virus-6.)

Singh et al also found significantly elevated levels of the IL-2, IL-12, and IFN-γ cytokines, indicators of an elevated inflammatory response, along with acute phase proteins, also indicative of inflammation.

These raised inflammation profiles are significant because subtle changes in the developing brains of autistic children have been found to be caused by autoimmune reactions leading to changes in the myelin sheath (a protective layer around nerves). This could lead to impairment in the brains of autistic children, resulting in problems with speech, communication, social action, and other neurological problems.

Partial Conclusion

At this point in their review, the authors point out that vaccines, which are used to “train” the immune system, lower its threshold of defense responses against development of infectious diseases. This leads to questions, including:

How can the immature, still developing immune and central nervous systems of a healthy child respond to such intense stimulation?
Can these immature systems respond with the same protective effect to so many different stimuli?
Do multi-antigen vaccine side effects change as compared to single-antigen vaccinations, and if so, how?

They go on to state:

[A] burgeoning body of evidence shows that immune molecules play integral roles in CNS development, affecting processes such as neurogenesis, neuronal migration, axon guidance, synaptic connectivity and synaptic plasticity. Despite the dogma that peripheral immune responses do not affect CNS function, substantial evidence points exactly to the contrary. Thus, it is not reasonable to assume that manipulation of the immune system through an increasing number of vaccinations during critical periods of brain development will not result in adverse neurodevelopmental outcomes.

“[I]t is not reasonable to assume that manipulation of the immune system through an increasing number of vaccinations during critical periods of brain development will not result in adverse neurodevelopmental outcomes.”

Clearly, these authors are stating that vaccines carry the potential to do tremendous harm.

These are the first two sections of the powerful Sienkiewicz, Kulak, Okurowska-Zawada, and Paszko-Patej review. The next article, No Historical Benefit in Vaccines: Polish Study, discusses the last two areas they covered: neurological symptoms following vaccination and a history of vaccines demonstrating little benefit.

Source:

Neurologic adverse events following vaccination; Progress in Health Sciences; Sienkiewicz D., Kulak W., Okurowska-Zawada B., Paszko-Patej G.

Tagged adverse events nervous system, adverse neurological outcomes, Kulak, Neurologic adverse events following vaccination, Okurowska-Zawada, Paszko-Patej, science, Sienkiewicz, toxicity of adjuvants, vaccine, vaccine damage, vaccines, vaccines adverse effects, vaccines adverse neurological outcomes, vaccines autism, vaccines autoimmune disorders, vaccines do irreparable harm, vaccines train the immune system

Antiperspirants: Cause of Breast Cancer?

Recent decades have brought enormous increases in breast cancer. Rather than look for a reason, most research has centered on finding treatments, though with little effect on outcomes. Philippa Darbre, though, has dedicated her efforts to finding if aluminum, a known human toxin that’s the basis for antiperspirants, could be the culprit.

by Heidi Stevenson

We live in the Age of Aluminum, a time that brings great convenience, but at the expense of enormous health damage. A likely case in point is the massive upsurge in breast cancer and fibrocystic disease, a condition of cyst formation in the breasts. Aluminum of the periodic table is the most likely cause of both these conditions.

Keele Conference participant Philippa Darbre has dedicated herself to researching this epidemic. She states:

Mortality results from breast tumour growth at metastatic sites rather than in the breast itself.

That is, breast tumors don’t kill, but their metastases do. The research she presented at February’s Keele Conference on the Biological Effects of Aluminum demonstrate that aluminum in the breast causes changes that can lead to deadly metastasis in breast cancer. She had, though, done a great deal of other research before broaching the issue of metastasis, including investigations of the relative amount of aluminum in cancerous and noncancerous breast tissue, the amount of aluminum found in breast fluid, dispersal of aluminum in breast tissue, and other issues related to aluminum and other metals in human health. Her focus is centered on aluminum in cosmetics, particularly antiperspirants.

Aluminum in Breast Tissue

Breast cancer is more common in the outer upper quadrant of the breast, which may be indicative of a connection with aluminum salts in antiperspirants. Drs. Darbre and Christopher Exley conducted a study to determine the amount of aluminum found in different parts of the breast.^[1] They measured the content of aluminum in the four breast quadrants of 17 women who had undergone mastectomies. The following graph shows the results:

Each bar represents 100% of the aluminum found in each woman, and is divided into four sections to indicate the percent of aluminum found in each quadrant. The bottom two sections show the percentage of aluminum found in the two outer quadrants, axilla and lateral. The graph shows that, on average, signifcantly more aluminum is stored in the outer area of women’s breasts.

This leads to two questions: Does aluminum cause breast cancer? and Is the concentration of aluminum in the outer quadrants of breasts a result of antiperspirants, whose active ingredient is an aluminum compound? This study clarifies, though, that aluminum found in antiperspirants must be considered a potential breast carcinogen.

Aluminum as an Estrogen Mimic

Many metals are estrogen mimics, making them potentially dangerous in the human body because of their ability to combine with estrogen receptors, thus replacing natural estrogen. Dr. Darbre did a study demonstrating that aluminum is also an estrogen mimic.

In the first part, she grew MCF7 breast cancer cells in the presence of estradiol (a type of estrogen) for 14 days. Then, she added one of different concentrations of two types of aluminum compounds used in antiperspirants, aluminum chloride and aluminum chlorhydrate and measured changes in the growth rate of the cancer cells.

Results are shown in the graph to the right. Both Al chloride and Al chlorhydrate interfere with breast cancer cell growth. Al chloride inhibited estradiol binding by 40% at 2.5 x molar excess (blue arrow) and 99.9% at 10 x molar excess (green circle). Al chlorhydrate inhibited estradiol binding by 64% at 5.0 x molar excess (red arrow) and 100% at 10 x molar excess (green circle).

The next experiment is similar, but rather than investigating interference with estradiol function, the needed amount of Al chloride and Al chlorhydrate to interfere with breast cancer cell growth was investigated. MCF7 cancer cells were grown in the lab and then concentrations of Al chloride or Al chlorhydrate were introduced. A concentration of 10^-3 mole of either Al chloride or Al chlorhydrate was required to cause disruption in cell growth. Therefore, MCF7 cells are resilient to high concentrations of these aluminum compounds.

The last part of this group of experiments demonstrated that aluminum, whether in the form of Al chloride or Al chlorhydrate, acts in the same manner as other estrogen-mimicking metals by increasing estrogen-regulated reporter gene expression, both in and out of estradiol’s presence.

Closing the Noose on Antiperspirants

These two studies by Dr. Darbre have shown:

Aluminum found in breast tissue of cancerous breasts is more prevalent in the outer breast, matching the most common location of breast cancer, the upper outer quadrant, the area where underarm antiperspirants are routinely used.
Aluminum in two compounds frequently found in antiperspirants acts as an estrogen mimic and interferes with estrogen’s function.

This doesn’t add up to proof positive that aluminum either causes breast cancer or that aluminum in antiperspirants does. It does, though, add up to prima facie evidence that it’s possible and that aluminum in antiperspirants should be put on trial. In the hope of stopping the breast cancer epidemic, the precautionary principle indicates that these products need to be investigated to find for sure whether there is a cause-and-effect connection.

Sadly, the health agencies that should be protecting us are not even looking at the risk. As a result, the science progresses slowly. Nonetheless, Philippa Darbre has taken the torch to carry this critical research forward. Do Antiperspirants Cause Breast Cancer? continues with more of her research that further tightens the noose on antiperspirants and their association with breast cysts and cancer.

Sources:

Aluminium in human breast tissue; Journal of Inorganic Biochemistry; Christopher Exley, Lisa M Charles, Lester Barr, Claire Martin, Anthony Polwart, Philippa D Darbre; doi:10.1016/j.jinorgbio.2007.06.005
Aluminium, antiperspirants and breast cancer; Journal of Inorganic Biochemistry; P.D. Darbre; doi:10.1016/j.jinorgbio.2005.06.001

Tagged aluminum antiperspirants, aluminum antiperspirants cancer, aluminum science, antiperspirants cancer, breast cancer cause, breast cancer etiology, breast cancer metastasis, breast cancer science, cancer outer quadrant breast, do antiperspirants cause cancer, keele conference, philippa darbre, science

Babies Used in Medical Vaccine Experiments (video)

How can anyone trust a medical system that has condoned medical experimentation on children without consent? Outside the Nuremburg rules? And to this day neither admits what it’s done nor aids those affected with gaining access to their records? Without even publishing the results? This is the system on which so many lives now depend and which demands sole authority over healthcare.

This video tells the true story of medical experimentation in Ireland and elsewhere in the world … including Australia, the US, Canada, the UK, and anywhere marginalized people can be found, such as Africa and India today. [33:19]

The Law Winks at Vaccines and Dead Babies

Tagged babies used for medical school autopsies, Babies Used in Medical Vaccine Experiments, big pharma, big pharma vaccine experiments, children used for medical expermentation, conventional medicine, medical experimentation, medical vaccine experimentation, modern medical system, modern medicine, science, vaccine, vaccines

NY to Give Vaccines to Minors Without Parental Consent

It’s illegal for children to vote. It’s illegal for children to choose to drink alcohol. It’s illegal for children to drive. It’s illegal for children to give consent for medical procedures. Yet, New York is planning to let children give consent for vaccinations, a potentially life-changing or destroying procedure, without parental consent?

by Christina England

On Monday, May 6, a new bill is to be brought to the New York State Assembly floor which will allow licensed physicians to administer preventative medical care for sexually transmitted diseases, including vaccines to minors without their parent’s permission.

This will include the vaccinations for hepatitis B and HPV.

The reasoning behind the bill is that section 2305 of the public health law currently permits a licensed physician or a staff physician to diagnose and treat a person under the age of 21 infected or exposed to a sexually transmissible disease without the consent or knowledge of the parents or guardians.

The bill states that the current law does not allow young people the same access to care as adults to prevent sexually transmissible diseases.

The bill states:

Teens should not be limited to access to care on a confidential basis after the fact, or after infection or contraction of a sexually transmissible disease. Teens should have access to confidential care before infection or contracting the sexually transmissible disease, to prevent disease or life-threatening illness such as cervical cancer and liver cancer.^[1]

When a bill of this nature becomes law, minors can be vaccinated without their parent’s permission, and vaccines for sexually transmitted diseases could also be able to be given to newborns without parental consent.

Don’t Let Your Child Be the Next Victim

The GreenMedInfo website has listed a total of 60 papers detailing the potential dangers of the Hep B vaccination.^[2]

However, if readers feel that researching all these papers is just a little overwhelming, then one of the strongest reminders of just how this vaccination can affect a newborn can be found on the Age of Autism website.^[3]

In 2009, the Age of Autism highlighted the tragic story of Ian.

Ian was born a completely healthy baby boy, although he did have one complication when he swallowed some of the meconium (stool) during delivery. After receiving treatment for breathing difficulties, it was not long before doctors stated that he was well enough to be discharged.

Despite the fact that Ian had a fever and was still fighting the side effects of swallowing meconium, Ian’s parents were asked to sign a form to allow them to administer the Hep B vaccination before their baby was released.

His mother takes up the story for the Age of Autism:

“After receiving the Hepatitis B shot these symptoms also appeared:

Platelets dropped from 248,000 to 131,000 – severe thrombocytopenia

Scalded-like rash appeared

Seizures noted

Irritable, crying non-stop

Stopped eating

Viral-like symptoms

His pictures speak a million words. Within 12 hours of his vaccination he had the rash, within 24 hours the severe thrombocytopenia set in, and then he was in a fatal state from then on. NO DOCTORS, NURSES, STAFF would even consider the vaccination as the source.

Ian lived for 47 days. My son suffered nearly his entire life. Look at the pictures. Imagine watching your child going through this pain and not being able to do anything. No one would listen to us…. we are just parents, what do we know? We didn’t leave his side for one moment. We lived at the hospital, literally. I couldn’t save my first born son, I couldn’t make the ‘ouchie’ better. I sat there day in and day out and watched this vaccination destroy his organ one at a time.”

The photos of baby Ian are probably the most horrific photos I have ever seen. They are stark reminder of just how dangerous this vaccination can be, a vaccination that the USA government, wants to administer to a minor without parental consent.

The Facts About This Dangerous Vaccination

Reports of adverse reactions show that as of mid-2012, the Vaccine Adverse Event Reporting System (VAERS) has received 119 reports of death following HPV vaccination, as well as:

894 reports of disability
517 life-threatening adverse events
9,889 emergency room visits
2,781 hospitalizations^[4]

According to Diane Harper, an international expert in HPV science, its vaccines, its clinical disease and treatment and the leading professional involved in the clinical trials of the HPV vaccinations, the duration for the effectiveness of the HPV vaccination Gardasil is just five years. In an interview with Marcia G. Yerman she states:

If the vaccinated person is not sexually active during the five years of its efficacy, then the vaccine has not protected her from disease (as we do not have evidence that Gardasil offers efficacy any longer than five years). Its faults include tiny antibody titers for all HPV types other than HPV 16; limited protection; limited duration of efficacy; and safety concerns (as outlined in my opening statement).

If this is correct, a child vaccinated at age eleven will not be protected on any level unless she becomes sexually active before the age of sixteen. Quite shocking, isn’t it?

One of the most damning facts that Ms. Harper revealed in her interview was when Ms. Yerman asked her this question:

Do you believe that the Gardasil vaccine, as it currently stands, could present more risks to a young girl or woman than the possibility of cervical cancer?

Ms. Harper answered:

Pap smears have never killed anyone. Pap smears are an effective screening tool to prevent cervical cancer. Pap smears alone prevent more cervical cancers than can the vaccines alone.

Gardasil is associated with serious adverse events, including death. If Gardasil is given to 11 year olds, and the vaccine does not last at least fifteen years, then there is no benefit – and only risk – for the young girl. Vaccinating will not reduce the population incidence of cervical cancer if the woman continues to get Pap screening throughout her life.

If a woman is never going to get Pap screening, then a HPV vaccine could offer her a better chance of not developing cervical cancer, and this protection may be valued by the woman as worth the small but real risks of serious adverse events.

On the other hand, the woman may not value the protection from Gardasil as being worth the risk knowing that 1) she is at low risk for a persistent HPV infection and 2) most precancers can be detected and treated successfully. It is entirely a personal value judgment.” ^[5]

I have also interviewed Ms. Harper and I also received similar answers.

How You Can Protect Your Child

Knowing that a leading expert has been prepared to state these facts, are you completely happy to have your rights as a parent taken away from you? If you are not, then the website truthaboutgardasil.com asks you to do the following:

Text of the bill.

Please contact any member of the New York State Assembly—or better yet, contact any of the bill’s sponsors, as seen in the link above to the text of the bill & then find their e-mail addresses—and let them know you oppose this bill! Also, if you are in New York, or are able to get there. A group of people affected by Gardasil will be making the trip to Albany this coming Monday, May 6, 2013 and would love to have more supporters join us! If you can’t make it, please send me a picture of the person affected, or even just their name so we can include them in the packet of information that will be handed out.

http://assembly.state.ny.us/leg/?default_fld=%0D%0A&bn=A497&term=2013&Summary=Y&Text=Y

If you have any questions, do not hesitate to contact us. moderator@truthaboutgardasil.org
or through Facebook at Marian Hollandsworth Greene ^[6]

Conclusion

Please act now if you want to have a say in what vaccinations your children receive. These vaccinations are not as safe as the US government wants you to believe they are. If you are still not convinced, please watch the following two videos on the HPV vaccines.

Gardasil. One More Girl

Cervarix Video

Sources:

1. http://assembly.state.ny.us/leg/?bn=A00497&term=…
2. http://www.greenmedinfo.com/anti-therapeutic-action/vaccination-hepatitis-b…
3. http://www.ageofautism.com/2009/02/managing-editors-note-below…
4. http://articles.mercola.com/sites/articles/archive/2013/01/24/hpv-vaccine-averting-cancer-unproven.aspx
5. http://www.huffingtonpost.com/marcia-g-yerman/an-interview-with-dr-dian_b_405472.html
6. http://truthaboutgardasil.org/new-york-is-trying-to-take-away-parental-rights-please-help/

Tagged children vaccination consent, conventional medicine, modern medicine, new york minor vaccination, new york vaccines, no parental consent, parental consent, politics, vaccination without parental consent, vaccine, vaccines

Welsh Measles Epidemic Was Faked

The Great Measles Epidemic of Wales—the one that’s being used to stampede sheeple into vaccine clinics for the MMR jab—never happened. Seriously! It was faked. The actual data from the Welsh government on cases of measles proves it.

by Heidi Stevenson,
with credit to Child Health Safety

Update: It is now 10 May 2013 and we have an updated report for you! As might be expected, the number of reported and confirmed measles cases has increased, though it also appears that this so-called epidemic is winding down.

The current number of reported cases for 1 January – 7 May is 1103. For some reason, the numbers for 2012 have been updated, with a total of 111 reported cases in November and December of 2012, for a total of 1214 reported cases during the recent outbreak. It should be noted that only 33 cases are reported for the first week of May, indicative of a strong slowing trend in the outbreak.

The number of confirmed cases lags the number of reported cases by a significant amount. Confirmed measles cases through 7 May is 279, and for November-December 2012 it was 13 (total of 18 for the year). In this measles outbreak, the total number of confirmed cases is now 292.

Why were the figures for 2012 changed? That’s a good question. Because of reader Tomas Kotrik’s inquiries (see comments below), Wales has added a statement to their website stating that confirmations from labs outside Wales are not included in these reports.

All measles patients are asked to give saliva samples, which are sent to labs for analysis. Therefore, the number of confirmed measles cases is salient to the severity of an outbreak. The number of reported cases is simply irrelevant. So, as the “Great Wales Measles Outbreak of 2013″ winds down, the total number of confirmed cases is only 292, only enough for a small fraction of GPs to have seen even a single case.Multiply the number ten times over, and you still wouldn’t have enough cases for every GP to see one. An epidemic? Hardly!

Updated Confirmed Data (through 7 May 2013)

Updated Reported Data (through 7 May 2013)

The news media have been chock-full of the “measles epidemic” in Wales, along with the young man whom it was claimed had died of it. He didn’t—and the epidemic never happened. The proof is in Wales’ own official statistics.

The young man who died during the proclaimed measles epidemic, Gareth Colfer-Williams, did not die from measles. But even those few reports that admitted he didn’t die of the disease went on to a discourse of how terrible measles is and how the “epidemic” was so horrible. In the BBC article linked, we’re informed that “83 people have needed hospital treatment for the illness.” That, though, cannot be true.

The fact is that, though 446 measles notifications were made between 1 January and 31 March of this year, those were merely reports. The reality is that only 26 cases were actually confirmed!

You may have noted that this faux measles epidemic started in November, and the figures for last year weren’t included. However, that doesn’t help make the case for an epidemic, or even come close to the claim that 83 people had to be hospitalized for measles. You see, the total number of confirmed measles cases in Wales for all of 2012 was 14. So, adding 14 for all of 2012 to 26 for the first three months of this year, we get a total of 40 confirmed cases of measles—less than half the falsely reported 83 hospitalizations!

Here’s the proof:

Wales Official Reported Measles Cases

Wales Official Confirmed Measles Cases

(You’ll need to scroll down to see the measles data.)

Admittedly, this doesn’t include the month of April—but with the presentation of this information, is it believable that the figures for April will make any difference? (Unless, of course, they’re simply faked—and who knows just how far these fear mongerers are willing to go?)

Keep in mind that measles is a reportable disease. That means any doctor who suspects someone has measles must report it. It does not mean that the person has the measles. When a disease is considered reportable and there’s supposedly an epidemic going on, doctors aren’t taking any chances with their licenses to practice. They’re reporting anything that bears the slightest resemblance to measles. However, as is obvious here, it doesn’t mean that the doctors have any skill at diagnosing it.

So what’s going on? It doesn’t take much to figure out that this has been nothing but a plan to fear monger people into rushing out for the MMR vaccine. Sadly, it’s worked. Far too many sheeple have been stampeded to the clinics. Baaa! Unfortunately, we can anticipate that it will also be used to promote forced vaccination.

Don’t ever forget that the Great Welsh Measles Epidemic of 2013 never happened. It was faked.

Tagged conventional medicine, measles epidemic fake, measles epidemic mmr vaccine, measles fear mongering, mmr vaccine, modern medicine, sheeple vaccinated for mmr, vaccine, vaccines, wales measles epidemic, welsh measles epidemic

Up to 100% Vaccinated Sheep Contract Chronic Neurological Syndrome: Study

Bluetongue can be a deadly disease of sheep. The standard approach to an outbreak is vaccination, vaccination, & more vaccination. A new study shows that these vaccines cause an autoimmune neurological disorder called ASIA—the same that vaccines can induce in humans. This study of Spanish sheep provides a frightening look into its frequency and severity. What does it mean in humans?

by Heidi Stevenson

The powers-that-be refuse to do studies comparing the health of vaccinated against unvaccinated children. However, Spanish animal pathology researcher, Lluís Luján, was concerned with serious health problems in sheep following mass vaccination to combat bluetongue, a viral disease spread by insects. So, a study was done to compare vaccinated versus unvaccinated sheep. The results should concern everyone. Though the percentages of affected sheep varied widely, as many as 100% of some flocks were devastated by the vaccine-induced disease called autoimmune/inflammatory syndrome induced by adjuvants (ASIA).

ASIA in Sheep

Researchers of the study, published in the journal Immunology Research, describe a syndrome that has an acute phase in only about one-half of one percent of sheep, from which most recover. However, a chronic phase develops later, and that is devasting, both in numbers and severity of illness. The economic impact is enormous. They describe it like this:

The chronic phase of the ovine ASIA syndrome is a more frequent event in our local conditions of ovine production and causes a neurological and cachectic process that has no parallel in ovine pathology, and it is a serious but unexplained concern for farmers and veterinarians. The chronic phase does not necessarily follow an acute episode, and it is
triggered by the combination of multiple alum adjuvantcontaining inoculations over the years and external stimuli.

Severity is generally related to stress. Conditions like cold weather, poor nutrition, and high levels of milk production result in significantly worse disease.

Sheep with Cachexia Resulting from ASIA

Frequency of chronic ASIA is about 50-70% of flocks, and up to 100% of the sheep within a flock. The syndrome runs through phases. It starts with “an excitatory period where affected animals show constant movement, abnormal behavior, restlessness and compulsive wool biting, resulting in animals with a very poor wool coat, a diffuse redness of the skin and thinning of the affected sheep.” During this time, the sheep exhibit a normal appetite or even an excessive one. However, this phase ends dramatically with sudden weakness, head tilting to one side, muscle tremors, and weight loss that moves toward cachexia.

Thickened Peripheral Nerves

In the terminal phase, the animals suffer from lack of response to stimuli, ataxia, and tetraplegia. They then fall into a stupor or coma, and die. They’re also known to have spontaneous abortions. They suffer from lesions of fat deposits, serous fat atrophy, ascites, hydrothorax, hydropericardium, and atrophy of skeletal muscles. Peripheral nerves become thickened in most animals. This can include primary nerves, like the sciatic, also.

Nerves in the spinal cord become necrotic. Most animals suffer from meningoencephalitis. Thickened peripheral nerves tend to be surrounded by fluid and mucous. Some demyelination and neuron loss was noted. About half the sheep were found to have membranous glomerulonephritis.

Stress conditions, such as winter lambing and cold weather, can trigger chronic ASIA. When there is an acute phase, it appears that it’s always followed by the chronic phase.

The Study

The researchers took 6 castrated 3-month old male lambs from an organic farm. They were divided into two groups of 3, one acting as controls remaining unvaccinated, and the other vaccinated according to the schedule on the right. The vaccines were for
a variety of ovine pathogens, including bluetongue (10 doses), Clostridium
(2 doses), Chlamydophila spp, Salmonella spp and abortive bacteria (2 doses). Each vaccine dose contained 4 mg aluminum salts and 0.2 mg thimerosal (thiomersal), for a total of 56 milligrams of aluminum and 2.8 mg of thimerosal. The controls were injected with a phosphate buffered saline solution.

The lambs were all kept together with their flock in their normal situation. The experiment ran from 28 June 2010 through 18 March 2011, which took the lambs through a winter cycle with their flock. On days 177, 254, and 261, pairs of control and vaccinated animals were killed for post mortem examination.

Weight of Vaccinated vs Unvaccinated Sheep

Weight Differences

The weight of the lambs was tracked and reported on the graph to the left. Almost immediately, the vaccinated lambs weighed less, though not by a lot. However, notice how the weights diverge with the weighing on the 177^th day, and the weight of the vaccinated sheep remains signigicantly lower. Winter temperatures started on day 164, causing stress that the vaccinated lambs were less able to withstand. Even after the weather started to warm again, you can see that, though both the controls and the vaccinated lambs gained weight, the vaccinated ones never made up the 8.5% difference lost during the stressful cold time.

Behavioral Differences

The researchers report that the vaccinated lambs exhibited different behaviors, including “an array of nervous clinical symptoms”, which worsened as time went on. They demonstrated periods of “depression with reluctance to move, light stupor and tendency to collapse when handling.” These periods of stupor and little movement alternated with “periods of excitement and abnormally stimulated behavior.” Note that this is much like what’s described for sheep in the early stages of ASIA syndrome.

Tissue Differences

The lambs were sacrificed and their tissues examined. The post mortem studies showed:

Accentuated depletion of fat deposits.
Mild hydroperitoneum and hydropericardium.
Small increase in the diameter of peripheral nerves.
Part of the spinal cord showed lesions indicative of early stage degeneration.

Aluminum and Mercury

The amount of mercury and aluminum in blood was examined in the 6 experimental animals, and also in 5 sheep with advanced ASIA disease:

The 5 sheep with advanced disease had 266.75 to 289.76 ng/mL of aluminum in their blood. No mercury was found.
Control animals had trace amounts of aluminum and no mercury in their blood.
Scanning electron microscopy found that the vaccinated animals had 8.445% aluminum weight, while controls had only 3.825% aluminum weight in their spinal cords.

Bluetongue Vaccine

The researchers’ primary concern is that the bluetongue vaccine was mandated for all sheep in Spain. After it was implemented, the effects on sheep flocks were devastating. Spanish sheep were inoculated four times per year for bluetongue—2 vaccines for different varieties of bluetongue, and then boosters for both each year. Each injection included aluminum adjuvant.

This study injected lambs with a more aggressive vaccination program in their first months of life. However, the total number of vaccines that each sheep receives year-on-year is greater than the lambs were given. They received a total of 14, while sheep in the flock normally received 4 standard vaccines each year, plus another 4 vaccines for bluetongue, reaching a total of 8 vaccines per year. So, any sheep past a year in age has already received more vaccinations than the seemingly over-vaccinated trial lambs.

Those 14 vaccines triggered an immediate acute ASIA response in the trial lambs, and experience has shown that it almost certainly would have turned into chronic ASIA, had the animals not been killed for the study.

As the results demonstrate, the toxic load of adjuvant aluminum that accumulates in blood and the central nervous system is very high over time, and its effect in the form of ASIA is devastating.

The question that must be addressed is whether the bluetongue vaccine makes sense in the face of ASIA syndrome. A natural part of the life cycle of these sheep involves the stress of winter cold. Its effect on the lower weight of vaccinated lambs is clear, and this study demonstrates that the stress will result in a significant number of sheep being lost—and in a particularly cruel manner.

So, let’s take a look at bluetongue. It’s caused by a virus that’s carried by some insects, such as midges. It can never infect humans, so protection for people is not necessary.

Bluetongue primarily affects sheep, then goats, and some cattle. In sheep, the disease can range from quite mild to deadly. It causes a nasal mucous discharge and fluid buildup around the mouth. In severe cases, the lips and tongue can be badly swollen, causing the tongue to protrude and become cyanotic—that is, turn blue, which gives bluetongue its name. The rest of the head and ears can develop edema. Excess blood can build up around the hooves, and if the animal is driven to keep moving, it can result in loss of the hoof. In severe cases, torticolis—twisting of the neck—can occur, along with vomiting, pneumonia, and conjunctivitis. The disease does need to be taken seriously.

However, death is not a given, and the death rate can be nil. The average mortality usually runs from 0-30 percent, though in particularly susceptible sheep, it can reach 100 percent. Only about 5% of cattle become ill from bluetongue, and death is rare.

Compare bluetongue with the devastating effects of its vaccine, and it doesn’t make much sense to be vaccinating against it. If a sheep has been infected and survives, then that sheep will be fine. However, when a sheep gets ASIA, then that sheep has a death sentence—and the reality is that it doesn’t appear to be any more likely that sheep will be harmfully infected with bluetongue than that they’ll be hit with ASIA from the vaccination.

Human Implications

ASIA is not simply a disease of sheep. Human versions of it are becoming better recognized, and their severity is much like that in sheep. Macrophagic myofasciitis (MMF) is one of them. Others may include Gulf War syndrome and other vaccine-related conditions. MMF is known to be induced by the vaccine adjuvant aluminum.

It’s certainly true that the fact aluminum in vaccines causes debilitating and deadly ASIA in sheep does not necessarily translate to humans. However, the evidence that aluminum adjuvants do cause ASIA-like syndromes in humans, coupled with how devastating ASIA is in sheep, not to mention how common, should trigger a regulatory response to eliminate aluminum from vaccines. Indeed, it ‘s truly impossible to find any reason other than selling out to explain why it’s still in them.

Source:

Autoimmune/autoinflammatory syndrome induced by adjuvants (ASIA syndrome) in commercial sheep; Immunology Research; Llui´s Luja´n • Marta Pe´rez • Eider Salazar • Neila A ´ lvarez • Marina Gimeno • Pedro Pinczowski • Silvia Irusta • Jesu´ s Santamari´a • Nerea Insausti • Yerzol Corte´s • Luis Figueras • Isabel Cuartielles • Miguel Vila • Enrique Fantova • Jose´ Luis Gracia Chapulle; DOI 10.1007/s12026-013-8404-0
Bluetongue, the Center for Food Security & Public Health

Tagged aluminum adjuvant asia, aluminum asia, asia autoimmuine, asia cachexia, asia neurology, asia syndrome, asia thickened nerves, autoimmune/inflammatory syndrome, bluetongue, bluetongue sheep, bluetongue vaccine, immunology research, Lluís Luján, macrophagic myofasciitis, mmf, ovine asia syndrome, science, sheep asia, sheep vaccine, vaccine, vaccine adjuvant asia, vaccine asia, vaccines

Vaccines’ Alum Adjuvant Path to Brain Found: Study

Newly published research by Keele Conference scientists shows that aluminum adjuvant in vaccines transfers to the brain. They have documented the path from injection site to the brain, and that once in the brain, it persists. Newborns, the elderly, and people with a certain genetic variation are particularly at risk.

by Heidi Stevenson

A new study adds a major link in the association of aluminum adjuvants in vaccines with neurological disorders. It demonstrates the pathway along which aluminum in vaccine adjuvants is transferred from the injection site to the brain, where it persists indefinitely. They have also identified a likely carrier and a means by which the process occurs.

The study is exhaustive, eliminating a wide array of options that might mitigate against their findings. They also focused on two groups to determine if their findings indicate that they’re at particular risk. They include newborns and people with a certain genetic variant. On top of that, the elderly may be at significantly higher risk for aluminum adjuvant-induced neurotoxic harm.

Please note that variant does not mean defective! We all have genes that are not like those of the majority. That does not indicate a defect, only a distinction. Viva la difference!

Reason for the Study

Nanoparticles are not as new in medicine as generally believed. The study states:

The use of nanomaterials in humans is not as contemporary as is recently portrayed. For decades, alum, a nanocrystalline compound formed of aluminum oxyhydroxide, has been the most commonly used adjuvant in vaccines.

In macrophagic myofasciitis (MMF), “alum-loaded macrophages [are] typically detected at sites of previous injections (up to >12 years later), resulting in a specific granuloma called macrophagic myofasciitis or MMF.” *

CCL2 is a cytokine that recruits monocytes, memory T cells, and dendritic cells to sites of inflammation. Earlier work has shown that 252 ASIA patients had an increase in circulating CCL2, along with a variation in the CCL2 gene. It’s also known that:

Danger signals from the CCL2 cytokine draw monocytes, which causes them to convert to macrophages before migrating to lymph nodes.
Dendritic cells (DCs) transfer antigens to a large network of distant dendritic cells, also known as antigen-presenting cells (APCs).
Alum injections are known to incite “significant changes linked to activation of the innate immune system in distant organs”.

These facts resulted in this study to detemine whether injected nanomaterials, most particularly alum, could be moved to distant organs through phagocytosis and CCL2 signaling.

The Experiments

Experiments were done on laboratory mice. A fluorescent surrogate for alum particles, fluorescent latex beads (FLBs), was used to assess movement and distribution of intramuscularly injected materials. Morin stain and particle induced X-ray emission (PIXE) were used to assess movement of alum hybrids, Al-Rho, which match the particle size of injected adjuvant aluminum. Using graphics from the study, I will attempt to explain what was done, though not all aspects of the study will be covered.

Granulomas

Standard C57BL/6 lab mice were injected in the tibialis anterior muscle (equivalent to a calf muscle in humans) with an alum-containing vaccine at a dose equivalent to that given to humans, with the amount calculated by an FDA standard. As is intended with an adjuvant, an intense localized inflammation ensued, which lasted until day 4 post-injection, when it stabilized. The local aluminum concentration decreased rapidly until day 4, remaining stable until day 21.

Aluminum-Laced Granuloma

Aluminum-loaded macrophages were found encased in granulomas from day 4 post-injection, as shown in this image. The left-hand image shows a photo of a granuloma, and the right-hand image is a PIXE graphic demonstrating the high aluminum load. (Yellow shows the greatest load.)

Signs of aluminum were found in the spleens and brains of these mice, as shown in these graphics:

Aluminum-Laced Spleen and Brain

Photographic images show spleen (left) and brain (right) tissues, with areas of PIXE imagery indicated with bright pink squares. The PIXE images showing aluminum loading are on the right side of each photo.

Aluminum Loading of Brain, Graphed Days 21 through 365, Plus Unvaccinated Controls

The graph on the left shows the percent of areas in the brains found to have aluminum spots. Notice the small number in unvaccinated mice. The numbers shown on days 21 and 365, one year post-vaccination, demonstrate that the aluminum does not dissipate. (The authors believe that the bar for day 180 is an anomaly, caused by either “interindividual variations in aluminum handling or to sampling problems related to variable proportions of grey and white matter in the randomly scanned areas”.

The aluminum that makes its way to the brain is persistent.

Movement of Alum Surrogates

FLB Movement by Macrophages, Hour 1 and Day 4

The image to the right shows the movement of FLBs by macrophages from the injection site to interstitial spaces in the muscle around it. Notice the bright red slash in the image to the left showing the fluorescing surrogates for alum, FLBs (fluorescent latex beads) one hour after injection. The right-hand image shows how they’ve been dispersed by macrophages four days later.

FLB Movement to Lymph Nodes, Hour 1 through Day 21

The image to the left is a graph of the number of cells loaded with FLBs (surrogate alum) found in popliteal (knee area) lymph nodes (P-DLN) and inguinal (groin) lymph nodes (I-DLN) at one hour, day 4, and day 21. Notice that movement of FLBs to lymph nodes is dramatic by day 4. Other images (not shown) document that this movement can be accounted for by inflammatory dendritic cells and macrophages that are derived from monocytes.

Another related part of this section of the experiment utilized injection of a compound known to inhibit FLB movement by phagocytes to popliteal lymph nodes. At day 4 movement to popliteal lymph nodes was down by 32%, and to inguinal lymph nodes by 69%. This treatment resulted in significant reduction of FLB in lymph nodes, further confirming that macrophages are the primary means of transportation to lymph nodes.

Graphs-of-Aluminum-found-in-Spleen-and-Blood

Graphs of FLBs found in Spleen and Blood

After moving to lymph nodes, the FLB alum surrogates moved to the spleen, as shown in the image to the right. The left-hand graph shows the number of FLBs in the spleen peaking at day 21 and decreasing to a little over half that number on day 90. Compare this with the previous graph, which shows FLBs leaving the lymph nodes between days 4 and 21. This increase in FLBs in the spleen between these same dates indicates that they’re moving from lymph nodes to the spleen.

The question then is: How does that happen? There is no connection between lymph nodes and the spleen. However, if you look at the graph on the right, you can see that FLBs are found in the blood, too. The authors surmise that the FLBs must travel in macrophages through the thoracic duct, the largest lymph vessel, which releases most of the body’s lymph into the blood system. Thus, macrophages carrying the alum-surrogate FLBs are transported from lymph nodes into the blood system, and then arrive in the spleen.

Graph of FLB Movement into Brain

The authors wrote, “Cerebral translocation of FLBs was delayed but relentless until the d90 endpoint in C57 mice,” and the graph to the left shows it. Only a small amount of the alum surrogate injected into the standard C57 lab mice reached the brain on day 4, but you can readily see how the pace picks up by day 21, and more than doubles again by day 90, the endpoint of the study on C57 mice.

The CX3CR1^GFP/+mice, indicated by the white bars, have an inserted gene that allows visualization of the microglia, which are a particular kind of macrophage found in the brain and spinal cord. As you can see, the FLBs were still increasing on day 180, the study’s endpoint for these mice.

FLBs in the Brainstem

The image to the right shows FLBs in the brainstem of a CX3CR1^GFP/+mouse. The inset is a photo of a stained slice of brain with their locations indicated by dots. The larger image is of an unstained section on day 21 showing the FLBs located mostly just under the pia membrane, i.e., on the surface of the brainstem.

Preventing FLBs from Transporting to Organs

Effect of I.V. Injections

Some C57 mice were given the FLB injection intravenously into the tail, rather than into the tibial anterior leg muscle. The result is shown in the graph on the left. Notice that hardly any FLBs ended up in the brain by either day 21 or 90. Clearly, little, if any, immune response resulting in macrophages engulfing FLBs is triggered by an intravenous injection.

Percent of FLB-Loaded Cells After Lymph Node Ablation

After ablating (destroying) popliteal and inguinal lymph nodes, the number of FLBs reaching the blood, spleen, and brain was reduced by 60-80%. The graph to the left shows the percentage of FLBs remaining compared to controls. The number that reached the spleen was only about 20% and the number reaching the brain was about 40%. This indicates that movement of FLBs by macrophages to local lymph nodes is part of the process for movement of most FLBs—and by analogy, most alum—from injection site to organs, including the brain.

Blood-Brain Barrier

FLBs with a Weak Blood-Brain-Barrier

One of the concerns in clinical procedures involving toxic elements on newborn babies is their weak blood-brain barrier. Mice with a leaky blood-brain barrier, called mdx, were used to see what happens in this situation. The graph to the right shows that, by day 21 the number of FLBs reaching the brain of an mdx mouse with a leaky blood-brain barrier is significantly greater than in a normal C57 mouse, and that this remains a problem over time.

Images from the muscles, spleen, and brain chimeric (genetically engineered) mice with a green fluorescing protein confirmed these findings.

Morin Stains Showing Al-Rho Particles

Confirmation: FLBs Correlate with Alum Adjuvant

The Morin stain for aluminum showed “Al-Rho particles were avidly phagocytosed after i.m. injection and formed intracellular agglomerates similar in size to the vaccine adjuvant”, as can be seen by the clusters in the “Vaccine” and “Al-Rho” images to the left. These images are similar to the first image of an aluminum-induced granuloma shown above.

The graphs below show Al-Rho movement from lymph nodes to spleen, and spleen to brain:

Al-Rho Movement to Lymph Nodes, Spleen, and Brain

The Al-Rho nanohybrid surrogates for alum showed up prominently in the popliteal and inguinal lymph nodes, which are nearest to the injection site, by day 4, as shown in the left-hand graph. Most have moved by day 21, showing up in the spleen, as you can see by the middle graph. By day 90, you can see that a significant number of Al-Rho have left the spleen. However, from the graph on the right, you can see that they’ve been moving from the spleen to the brain. The movement is strikingly similar to that of the FLBs.

Effect of CCL2 Cytokines in Alum Distribution

The CCL2 cytokine is suspected to be at the center of some ASIA disorders because research has shown that ASIA patients have high amounts in their blood, along with a genetic variant related to CCL2. Mice bred to be deficient in the CCL2 cytokine were used to see what effect they have on distribution of the alum surrogate, Al-Rho, to lymph nodes and organs.

Movement of Al-Rho in CCL2-Deficient Mice

In the left-hand graph of the image to the right, you can see that there is significantly less movement of Al-Rho from the site of an intramuscular injection to organs. Compared with controls, 65% the amount of Al-Rho is found in the nearest lymph node at day 4, and even less, 34%, gets to the inguinal lymph node. On day 21, only 15% as much is found in the blood, 29% in the spleen, and only 18% as much Al-Rho is found in the brains of CCL2-deficient mice compared to controls.

The graph to the right of “CCL2 Deficient Mice” shows the amount of Al-Rho found in the same kind of CCL2-deficient mice after they’ve been injected with rCCL2, a recombinant form of CCL2 that was used because no other version is available. The results are dramatically different. First, note that the y-axis scale is dramatically different, running up to 600%, instead of only 80%. By day 21, the rCCL2 injections increased distribution of Al-Rho to the blood by 274%, the spleen by 180%, and the brain by 341% as compared to controls.

These experiments, along with others not described here, document that more CCL2 cytokines—which draw monocytes and dendritic cells to the site of inflammation—than normal are directly associated with the distribution of alum to the brain.

Implications

This study has demonstrated that aluminum adjuvant particles can be transported to the brain through CCL2 signaling that brings monocytes to the site of vaccine administration. The route is shown to be:

First to the nearest lymph nodes.
Through the lymph system’s thoracic duct.
Through the thoracic duct into the bloodstream.
Through the blood to the spleen and possibly partially directly to the brain.
From the spleen to the brain.

This process appears to occur in all mice, though at a slow rate. However, in mice with high levels of CCL2 cytokines, the process is speeded up dramatically. The study also documented that an inefficient blood-brain barrier, as is found in the youngest babies, allows significantly more alum through to the brain.

The toxic potential of aluminum is high. This study has demonstrated that injecting alum adjuvants with vaccines results in transferrence to the brain, where it persists. Most people have a high tolerance to alum. However, there are limits in anyone. In the case of people with high levels of CCL2—such as those with a genetic variant leading to high CCL2 levels, as found in ASIA patients—even small amounts of injected alum can be disastrous.

Consideration needs also to be given to the association of aluminum in the brain with Alzheimer’s disease, which is afflicting an accelerating percentage of people as they age.

As the study’s authors point out:

It is likely that good tolerance to alum may be challenged by a variety of factors including overimmunization, BBB immaturity, individual susceptibility factors, and aging that may be associated with both subtle BBB [blood-brain barrier] alterations and a progressiveincrease of CCL2 production.

With the rapidly increasing number of vaccinations recommended, and even mandated, in schedules, along with the increasing number of people suffering from chronic neurological and autoimmune disorders, surely it’s past time to sound the alarm. At the very least, serious research to investigate these risks must be launched.

The researchers involved with the Keele Conference are doing valuable research aimed at identifying and mitigating these risks. It’s long past time for the agencies supposedly concerned with public health to follow these leaders of science.

Three of the study’s authors, Christopher Exley, Romain Gherardi, and François-Jérôme Authier, attended the 10^thKeele Conference titled, “Illuminating and Elucidating Aluminium’s Exposome: From Geochemistry to Neurochemistry, From Microbe to Man”:

Dr. Exley was the conference administrator and has published a large number of studies on the topic, not to mention being involved in several of the studies and references of this conference.
Dr. Gherardi presented his latest work on macrophagic myofasciitis (MMF), a new vaccine-associated disease, indicating that the cytokine CCL2 is not simply a biomarker of MMF inflammation, but is also associated with its pathophysiology. He suggested that CCL2 should be investigated in relation to other autoimmune/inflammatory syndromes (ASIA).
Dr. Authier reported on a new non-invasive method to diagnose MMF, which has hitherto been diagnosable only via muscle biopsy. He also suggested that MMF is probably more prevalent than currently realized.

* Unattributed quotations are from the study.

Source:

Slow CCL2-dependent translocation of biopersistent particles from muscle to brain, Biomed Central Medicine; Zakir Khan, Christophe Combadière, François-Jérôme Authier, Valérie Itier, François Lux, Christopher Exley, Meriem Mahrouf-Yorgov, Xavier Decrouy, Philippe Moretto, Olivier Tillement, Romain K Gherardi, and Josette Cadusseau; doi:10.1186/1741-7015-11-99

Tagged adjuvant brain, adjuvant spleen, al-rho, alum adjuvant, alum brain, alum vaccine adjuvant, aluminum brain, asia vaccine, biomed central, ccl2, ccl2 alum, ccl2 cytokine, ccl2 vaccines, dendrite, dendrite vaccine, flb, fluorescent latex bead, granuloma adjuvant, granuloma alum, granuloma vaccine, macrophage, macrophage vaccine, monocyte, monocyte vaccine, nano adjuvant, nano aluminum, nano vaccine, phagocytosis, Slow CCL2-dependent translocation of biopersistent particles from muscle to brain, surrogate adjuvant, vaccine adjuvant

Oil from Petroleum Used as Vaccine Adjuvant

Petroleum forms the basis of one of the most devastating adjuvants in existence. Historically, it was considered too dangerous for use in vaccines, but it’s making a comeback in this new age of recombinant DNA vaccine development. Of course, the public relations mantra is that it’s safe—so why is it kept quiet?

by Heidi Stevenson

The current hype in a new vaccine against foot-and-mouth disease is all about how the new ones are safer. But are they? The clue is in what isn’t discussed: the adjuvants. PLoS reports that a UK government and Big Pharma funded study has found a new approach, using recombinant DNA technology, to create empty capsids, that is, empty virus protein shells, to use as antigens against foot-and-mouth disease.^[1]The hollow antigens would be grown inside insects.

Antigens and Adjuvants

Only the surface of an antigen is required to show the immune system how to create antibodies. This method of vaccine development makes it impossible to spread the infection, since none of the active part of a virus is included. It also means that vaccine antigens can be made both rapidly and relatively cheaply.

There’s one big disadvantage, though. The immune system isn’t all that dumb. It does appear to be capable of distinguishing between genuine and fake threats. So, something is needed to trick it.

These synthetic antigens aren’t able to trigger much of an immune response when injected. Therefore, an adjuvant is required, and it must be particularly strong for the vaccine to trigger a strong enough immune response to result in antibodies to the antigen.

The problem is that all adjuvants are, by definition, toxic. Their function is to fire up the body’s immune system. Try this analogy:

Photo by Njambi Ndiba

Picture a tiger in a cage, one that’s well-fed, content, and has spent his entire life in it. The audience wants a show and the zookeepers want to give it. So they toss some chicken skins in his cage, hoping he’ll get excited, and start growling and shredding the skins.

But they fail to interest him. He’s used to a certain amount of aggravation. Kids hooting don’t faze him at all. People tossing odds and ends of things into his cage … oh well, he’s gotten used to it. So, the zookeepers—having no moral sense about the animals under their care—spray the tiger with pepper spray.

Now, that gets him riled up! He roars and looks for something to attack. And what does he find? Those chicken skins. So he puts on a great show, fussing and fuming and shredding those skins to bits.

The pepper spray is equivalent to a vaccine adjuvant. Had the zookeepers tossed live chickens into the cage, that tiger would have gone after them and put on a great show. But chicken skins just weren’t interesting enough.

Empty capsids are to the immune system much like chicken skins are to that tiger. You can inject and inject them, but the immune system won’t care. So, you need to toss something really aggravating into the mix. That something is a strong adjuvant.

And that’s exactly what this vaccine will require. According to the PLoS study, the adjuvant proposed for use is called Seppic 206B. The manufacturer’s website defines it as:

Ready-to-use oily vaccine adjuvant for water-in-oil-in-water (W/O/W) emulsion

Based on high-grade injectable mineral oil^[2]

Mineral oil is made from petroleum. It is the same oil that’s the basis of Freund’s adjuvant, which is used to create autoimmune disorders in laboratory animals by injection. Shortly after discovery, they were banned from use in humans because they were recognized as too dangerous.

And this is what is now being praised as safe. The publicity gives the impression that the vaccine itself is safe, but careful reading shows that the safety element is associated only with the manufacturing process—the fact that no live virus is used, so there’s no chance of accidental infection or escape. It has nothing to do with the safety of injecting it.

Animal Abuse

The initial goal of this new technology is to produce a new vaccine against hoof-and-mouth disease so that it can be used on a mass scale in livestock. The plan, obviously, is to use the same kind of adjuvant that’s sold to science labs around the world for the purpose of creating severe autoimmune disorders like rheumatoid arthritis in lab animals.

As long as their pain from these horrible diseases isn’t obvious, who’s going to know how the animals suffer? You know that no one’s going to be watching to see if they’re harmed. One of the most difficult things to deal with in an autoimmune disorder is that they often don’t show on the outside. Sufferers of myalgic encephalitis (chronic fatigue syndrome) have routinely been accused of malingering simply because their misery showed only on their faces.

In the case of animals raised for food … well, these won’t be healthy beasts, and it isn’t healthy to eat meat from sick animals. In the case of animals raised for dairy, their milk would likely contain the inflammatory agents that result from an autoimmune disorder. You have to wonder at that. Will they redefine the “safe” levels of inflammation markers in milk, since they usually won’t find pathogens associated with it? Or just pasteurize it more? That process already exists in long shelf life milk that doesn’t require refrigeration.

The Real Goal

The study’s authors state:

… the approach we have demonstrated here may be applicable across a wide range of human and animal picornaviruses, including polioviruses and coxsackieviruses.^[1] [Emphasis mine.]

It is not the intention to use this technology only on animals. The goal is to develop vaccines for humans. These vaccines, by their very nature, will require very strong adjuvants.

The nature of any adjuvant is to be toxic. That’s what makes them work. The weaker the antigen, the stronger the adjuvant must be. The new technology utilizes an adjuvant that’s known to be extremely toxic, so much so that it’s used to produce autoimmune disorders in lab animals.

This mineral oil adjuvant is already in use on farm animals. Aside from the incredible abuse that factory farming heaps on those poor beasts, how many of them live in constant pain simply because of the vaccinations given? But these animals are kept out of sight—and out of sight does tend to mean out of mind. After all, most people still buy their meat in supermarkets, which are the mainstay of these industrialized farms.

The real goal is to bring these mineral oil based adjuvants to humans. These, and others like squalene, which is responsible for the recent mass outbreak of debilitating narcolepsy from the swine flu vaccine, will be used so that Big Pharma can produce vaccines quickly and cheaply.

We’re living in an era of mass vaccination. The results are showing up in our children. The average child is now sick. The countries with the highest vaccination rates have the highest child mortality rates. Rather than do legitimate studies to identify the reasons for it, we see junk study after pseudo study trying to convince us that vaccines are just fine. Meanwhile, even with strong evidence of vaccination harm, the push is on to produce more and more, faster and faster.

And the lives that are devasted? Oh well, that’s too bad … and anyway, it’s mere coincidence when a child gets autism or arthritis or asthma or bullous pemphigoid or cancer or diabetes or eczema or …

Note: Study funders were the Wellcome Trust, a foundation started by the company now called GlaxoSmithKline; DEFRA, UK agency that controls animal vaccination and promotes it; Medical Research Council, an agency funded by the taxpayers.

Five of the nine researchers have deep ties to the vaccine industry, including a lead researcher who’s a Jenner Investigator, two students supported by the Wellcome Trust, and two supported by the Medical Research Council.

Sources:

Rational Engineering of Recombinant Picornavirus Capsids to Produce Safe, Protective Vaccine Antigen. PLoS Pathog; Porta C, Kotecha A, Burman A, Jackson T, Ren J, et al. (2013) 9(3): e1003255. doi:10.1371/journal.ppat.1003255
Montanide ISA 206 VG
New foot-and-mouth vaccine is safer and cheaper to produce
New hope for safer, more stable foot and mouth disease vaccine
Scientists develop new foot and mouth vaccine
Montanide ISA 206 VG
Aspects of the role of mineral oil as immunological adjuvants in rheumatoid arthritis
Autoimmunity induced by adjuvant hydrocarbon oil components of vaccine

Tagged adjuvant toxic, chronic disease vaccines, empty capsids vaccine, freund’s adjuvant, immune system adjuvants, immune system antibodies, immune system antigens, immune system vaccines, junk science, junk study, mineral oil adjuvant, mineral oil petroleum, oil adjuvant, petroleum adjuvant, pseudo study, pseudo-science, pseudoscience, recombinant dna technology, science, vaccine, vaccine adjuvant, vaccines